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For instance, upregulating GPX4 in ferroptosis-sensitive chondrocytes, restoring antioxidant defenses in inflammatory synovial macrophages, or rebalancing iron export in subchondral osteoblasts may prevent tissue-specific degeneration
For a long time, scientists believe that chronic neuroinflammation is crucial in cognitive impairment, and that GLP-1RAs protect synaptic and learning functions from neuroinflammation (119, 120)
Selective and membrane-permeablesmall molecule inhibitors of nicotinamide N-methyltransferase reverse high fat diet-induced obesity in mice
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